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The apoptotic effect of 1S1Acetoxychavicol Acetate ACA enhanced by inhibition of noncanonical autophagy in human nonsmall cell lung cancer cells
Description The apoptotic effect of 1S1Acetoxychavicol Acetate ACA enhanced by inhibition of noncanonical autophagy in human nonsmall cell lung cancer cells.
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The apoptotic effect of 1’S-1’-Acetoxychavicol Acetate ~ Autophagy plays a role in deciding the fate of cells by inducing either survival or death. 1’S-1-acetoxychavicol acetate (ACA) is a phenylpropanoid isolated from rhizomes of Alpinia conchigera and has been reported previously on its apoptotic effects on various cancers. However, the effect of ACA on autophagy remains ambiguous. The aims of this study were to investigate the autophagy .
(PDF) The apoptotic effect of 1’S-1’-Acetoxychavicol ~ The apoptotic effect of 1’S-1’-Acetoxychavicol Acetate (ACA) enhanced by inhibition of non-canonical autophagy in human non-small cell lung cancer cells February 2017 PLoS ONE 12(2):e0171329
The apoptotic effect of 1′S-1′-Acetoxychavicol Acetate ~ The apoptotic effect of 1′S-1′-Acetoxychavicol Acetate (ACA) enhanced by inhibition of non-canonical autophagy in human non-small cell lung cancer cells Sophia P.M. Sok, Norhafiza M. Arshad, Mohamad Nurul Azmi, Khalijah Awang, Bulent Ozpolat , Noor Hasima Nagoor
The apoptotic effect of 1’S-1’-Acetoxychavicol Acetate ~ apoptotic effect of 1’S-1’-Acetoxychavicol Acetate (ACA) enhanced by inhibition of non-canonical autophagy in human non-small cell lung cancer cells. PLoS ONE 12(2): e0171329. doi:10.1371/ journal.pone.0171329 Editor: Sujit Kumar Bhutia, National Institute of technology Rourkela, INDIA Received: July 22, 2016 Accepted: January 18, 2017
The Apoptotic Effect of 1’S-1’-Acetoxychavicol Acetate ~ Abstract: 1’-(S)-1’-Acetoxychavicol acetate (ACA) isolated from the Malaysian ethno-medicinal plant Alpinia conchigera Griff. was investigated for its potential as an anticancer drug. In this communication, we describe the cytotoxic and apoptotic properties of ACA on five human tumour cell lines.
The Apoptotic Effect of 1’S-1’-Acetoxychavicol Acetate ~ 1’-(S)-1’-Acetoxychavicol acetate (ACA) isolated from the Malaysian ethno-medicinal plant Alpinia conchigera Griff. was investigated for its potential as an anticancer drug. In this communication, we describe the cytotoxic and apoptotic properties of ACA on five human tumour cell lines. Data from MTT cell viability assays indicated that ACA induced both time- and dose-dependent .
The apoptotic effect of 1'S-1'-Acetoxychavicol Acetate ~ The apoptotic effect of 1'S-1'-Acetoxychavicol Acetate (ACA) enhanced by inhibition of non-canonical autophagy in human non-small cell lung cancer cells Sok, Sophia P. M. Arshad, Norhafiza M.
The Apoptotic Effect of 1 ' S-1 '-Acetoxychavicol Acetate ~ 1'-(S)-1'-Acetoxychavicol acetate (ACA) isolated from the Malaysian ethnomedicinal plant Alpinia conchigera Griff. was investigated for its potential as an anticancer drug. In this communication, we describe the cytotoxic and apoptotic
The apoptotic effect of 1’S-1’-Acetoxychavicol Acetate ~ The apoptotic effect of 1’S-1’-Acetoxychavicol Acetate (ACA) enhanced by inhibition of non-canonical autophagy in human non-small cell lung cancer cells By Sophia P. M. Sok, Norhafiza M. Arshad, Mohamad Nurul Azmi, Khalijah Awang, Bulent Ozpolat and Noor Hasima Nagoor
Pro-apoptotic effects of 1′-acetoxychavicol acetate in ~ 1′-Acetoxychavicol acetate (ACA) is a ginger-derived compound that has antitumorigenic properties in various cancer animal models and human cancer cell lines. One of the early studies with this compound showed its inhibitory effects on tumor-promoter-induced Epstein-Barr virus activation in vitro ( Kondo et al., 1993 ).
Apoptotic Pathways: Cell ~ Apoptotic cell death is found throughout the animal kingdom, and it culminates in the execution, packaging, and disposal of the dying cells. Several recent advances have contributed to an emerging (and evolving) view of how apoptosis proceeds once initiated, to the point that we can speculate on a pathway for this remarkable process.
Inhibitory effects of persistent apoptotic cells on ~ Introduction. Cell death occurs normally in all tissues via well-known physiological mechanisms, the most widely studied of which is apoptosis. A key feature of apoptosis is its non-phlogistic nature: apoptotic cells are rapidly removed by phagocytes in vivo without causing inflammatory responses or damage to neighboring cells.
Apoptotic Pathways: Cell ~ Nearly all physiological cell deaths in animals proceed by the process of apoptosis, during which the dying cells vanish without a trace, silently cleared without any accompanying inflammatory response. Like the last scene of a noir mystery, we can now line up the various molecular suspects in this cell death. Here, we will examine recent knockouts and other studies that shed light on the .
Inhibition of Apoptosis / Cell Signaling Technology ~ Cell survival requires the active inhibition of apoptosis, which is accomplished by inhibiting the expression of pro-apoptotic factors as well as promoting the expression of anti-apoptotic factors. The PI3K pathway, activated by many survival factors, leads to the activation of Akt, an important player in survival signaling.
Apoptosis - Wikipedia ~ Apoptosis (from Ancient Greek ἀπόπτωσις, apóptōsis, "falling off") is a form of programmed cell death that occurs in multicellular organisms. Biochemical events lead to characteristic cell changes and death.These changes include blebbing, cell shrinkage, nuclear fragmentation, chromatin condensation, chromosomal DNA fragmentation, and global [vague] mRNA decay.
Different apoptotic mechanisms are involved in the ~ When cells were exposed for 32 h to 60 μM 7β-OHsito or to 30 μM 7β-OHchol, both compounds caused 50% growth inhibition. Cells treated with 7β-OHsito showed enhanced caspase-9 and -3 .
Evaluation of anti-apoptotic activity of different dietary ~ Also, the anti-apoptotic effect of antioxidants on cancerous cells (RCC-26) is quite evident. The protective effect of antioxidants over cells against ROS has been studied. Antioxidants prevent radical (especially hydroxyl radical generated by H 2 O 2) mediated cell damage and indirectly suppress apoptosis. Thus, poly-phenolic antioxidants show .
Apoptosis drives cancer cells proliferate and metastasize ~ The prolonged cell cycle of cancer cells is contrary to many people's beliefs. What cancer affects on the duration of cell cycle is a very tricky question for cancer researchers. An overwhelming majority of people have the wrong conception that ‘the duration of cell cycle of cancer turns short’.
Apoptosis - an overview / ScienceDirect Topics ~ Apoptotic cells display a range of signals to attract phagocytes, including changes in cell surface molecules such as sugars, lipids, and proteins. Although phagocytic recognition of apoptotic cells is a critical process and an active field of research, details of this final event in apoptosis are beyond the scope of this review.
Mechanism of apoptotic effects induced selectively by ~ The DNA ladder formation, characteristic of ultrastructural appearance of apoptotic cells, and the augmentation of apoptotic sub-G1 population indicate that apoptosis is a major underlying process induced by UDCA. Cell proliferation is defined as the increase in cell number resulting from completion of the cell cycle.
Anti-apoptotic signaling by hepatocyte growth factor/Met ~ In agreement with this possibility, LY294002 and PD098059 had an additive, but not synergistic, effect on cell viability and caspase-3 activation and abolished the anti-apoptotic effect of HGF/Met signaling. In conclusion, this study implicates apoptosis inhibition via PI3-kinase/Akt and MAPK as a critical component of Met oncogenesis.
Antiapoptotic - an overview / ScienceDirect Topics ~ In colon cancer cells, induction of G2/M arrest was mediated by action of MAP kinases like ERK, JNK, and p38 [88,89]. SFN also induced cell cycle arrest in Nrf2 −/− cells by inhibition of HDAC activity. This indicates that the anticancer effect of SFN is independent of Nrf2 [90,91].
Apoptosis: A review of pro‐apoptotic and anti‐apoptotic ~ A feature characteristic of cancer cells is the uncoupling of cell division and cell death; cells that should have died were not properly signaled to do so. 3 Oncologic studies are investigating the enhancement of apoptosis to arrest the growth of tumor cells. One mechanism through which normal cellular numbers are maintained is through p53 and .
The Apoptotic Effects and Synergistic - Cancer Research ~ This study deals with the apoptotic effect exerted on human retinoblastoma Y79 cells by both sodium butyrate and an inhibitor of 26S proteasome [z-Leu-Leu-Leu-CHO (MG132)] and their synergistic effect. Exposure to sodium butyrate (1–4 mm) induced an accumulation of cells in the G2-M phase that was already visible after 24 h of treatment, when morphological and biochemical signs of apoptosis .